Welcome from the Director

Welcome to the C.S. Mott Center for Human Growth and Development, an internationally known research center established to promote research training relating to women's and children's health with focus on reproductive biology, immunology, oncology, toxicology and prenatal medicine. Our scientists are shaping the understanding of how human being's health is molded even before the fetus begins to grow. We integrate basic, translational and clinical research with the purpose of improving women's health. We have the infrastructure in place to actively contribute to the community as the leading translational center for women's health.
The Mott Center has championed a life-span perspective to reproductive health and an ecological approach to growth, development and well-being. We recognize our responsibility to engage our unique urban environment as we approach reproductive life sciences from bench to bedside and into the community. We are about breaking down barriers and unifying the necessary technologies with expert physician-scientists and the patients we serve.
Read our full mission.
Research
News and announcements
- Evaluating the epigenetic age of sperm to predict pregnancy outcomes
- Mott Center continues 50th anniversary celebration year
- Zhibing Zhang lab wins $2.4 million from National Institute of Child Health and Human Development to combat male infertility
- School of Medicine professor discusses fertility's new frontier at Wall Street Journal's Future of Everything Festival
- C.S. Mott Center for Human Growth and Development will hold open house June 9
Publications
- MNRR1 is a driver of ovarian cancer progression
- Generation of Stable Epithelial–Mesenchymal Hybrid Cancer Cells with Tumorigenic Potential
- Leukocyte immunoglobulin-like receptor subfamily B: A novel immune checkpoint molecule at the maternal-fetal interface
- Exposure to a mixture of per-and polyfluoroalkyl substances modulates pulmonary expression of ACE2 and circulating hormones and cytokines
- Decorin promotes decidual M1-like macrophage polarization via mitochondrial dysfunction resulting in recurrent pregnancy loss